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眼輪筋反射の第2コンポーネントの反射経路についての臨床的検討
https://kindai.repo.nii.ac.jp/records/2001600
https://kindai.repo.nii.ac.jp/records/2001600120433ab-66e9-4aeb-93cb-63bce59b3389
| 名前 / ファイル | ライセンス | アクション |
|---|---|---|
AN00063584-19941225-0445.pdf
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| Item type | ☆紀要論文 / Departmental Bulletin Paper(1) | |||||||||
|---|---|---|---|---|---|---|---|---|---|---|
| 公開日 | 2024-07-19 | |||||||||
| タイトル | ||||||||||
| タイトル | 眼輪筋反射の第2コンポーネントの反射経路についての臨床的検討 | |||||||||
| 言語 | ja | |||||||||
| タイトル | ||||||||||
| タイトル | The study in search for the pathway of the blink reflex R2 component with regard to the central nervous system lesions | |||||||||
| 言語 | en | |||||||||
| 著者 |
内山, 卓也
× 内山, 卓也
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| 言語 | ||||||||||
| 言語 | jpn | |||||||||
| キーワード | ||||||||||
| 主題 | blink reflex, the late component (R2), hemispheric lesion, short latency somatosensory evoked potential (SSEP) | |||||||||
| 資源タイプ | ||||||||||
| 資源タイプ識別子 | http://purl.org/coar/resource_type/c_6501 | |||||||||
| 資源タイプ | departmental bulletin paper | |||||||||
| 版 | ||||||||||
| 出版タイプ | AM | |||||||||
| 出版タイプResource | http://purl.org/coar/version/c_ab4af688f83e57aa | |||||||||
| 出版者 名前 | ||||||||||
| 出版者 | 近畿大学医学会 | |||||||||
| 言語 | ja | |||||||||
| 書誌情報 |
ja : 近畿大学医学雑誌 en : Medical Journal of Kinki University 巻 19, 号 4, p. 445-464, 発行日 1994-12-25 |
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| ISSN | ||||||||||
| 収録物識別子タイプ | PISSN | |||||||||
| 収録物識別子 | 03858367 | |||||||||
| 内容記述 | ||||||||||
| 内容記述タイプ | Abstract | |||||||||
| 内容記述 | The blink reflex usually presents two components; R1 and R2. The early reflex component, R1, has a latency range in 8-12 ms. and arises ipsilaterally to the stimulated supraorbital nerve. The late bilateral component, R2, shows a latency range in 25-40 ms.. The difference in latency between right and left R1 dose not exceed 1.5 ms. in normal subject. And that between right and left R2 dose not exceed about 8 ms.. The blink reflex especially R2's (direct and consensual) are often absent or depressed in amplitude in the case of supratentrial and infratentrial lesions. Electrically elicited blink reflexes investigated in this study totaled 57, including 20 normal subjects, 6 cases of thalamic lesion (4 cases were hemorrhage, 2 cases were tumors), 4 cases of perithalamic lesion (3 cases were hemorrhage, 1 case was infarction), 8 cases of putaminal lesion (5 cases were hemorrhage, 3 cases were infarction), 11 cases of frontal lobe lesion (6 cases were tumors, 4 cases were hemorrhage, and 1 case was infarction), 4 cases of temporal lobe lesion (3 cases were tumors and 1 case was infarction), and 4 cases of chronic subdural hematoma. CT or/and MRI examination and SSEP recording were also performed in parallel with this study. The late response R2 evoked by electrical stimulation to the supraorbital nerve, contralateral side to the side of hemispheric lesion was absent or suppressed in 25 out of 37 cases. An abnormal R2 was observed in this study in 70% of the thalamic and perithalamic lesions, 75% of the putaminal lesions, 82% of the frontal lobe lesions, 75% of the temporal lobe lesions, and 0% of the chronic subdural hematomas. Thalamus, trigeminal lemniscus as well as bulubar lateral reticular formation of lower brain stem may play important roles in the afferent pathway of the reflex arc of R2. Corticobulbar fiber via pyramidal tract from contralateral lower postcentral region have been advocated by Ongerboer de Visser to play an important role in the efferent pathway of the reflex arc of R2. Furthermore, in our investigation, the corticospinal tract and corticotegmentales fiber might be considered to participate in the efferent pathway of the reflex arc of R2. The abnormal patterns of R2, especially afferent and efferent block, may indicate decreased excitability of the bulbar lateral reticular formation of lower brain stem. | |||||||||
| 言語 | en | |||||||||
| 内容記述 | ||||||||||
| 内容記述タイプ | Other | |||||||||
| 内容記述 | 本文データはCiNiiから複製したものである。 | |||||||||
| 言語 | ja | |||||||||
